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One possibility is that diminished expression of Slc7a11 in Nrf2-null mice limits the supply of intracellular cystine, which is reduced to cysteine for protein synthesis [164], and that lower levels of cysteine may compromise the correct folding of membrane proteins and secretory proteins within the ER Another possibility is that the diminished availability of GSH, or a relative inability to reduce GSSG, compromises the ability of cells to fold proteins into their appropriate native tertiary structures